Название продукции:9-Bromo-5,6-dihydro-2-iodo-imidazo[1,2-d][1,4]benzoxazepine
IUPAC Name:12-bromo-4-iodo-9-oxa-3,6-diazatricyclo[8.4.0.0²,⁶]tetradeca-1(14),2,4,10,12-pentaene
- CAS:1282516-69-1
- Молекулярная формула:C11H8BrIN2O
- Чистота:95%+
- Номер в каталоге:CM152652
- Молекулярная масса:391.01
Только для использования в НИОКР..
Информация о продукции
- Номер CAS:1282516-69-1
- Молекулярная формула:C11H8BrIN2O
- Точка плавления:-
- Smiles-код:IC1=CN2CCOC3=CC(Br)=CC=C3C2=N1
- Плотность:
- Номер в каталоге:CM152652
- Молекулярная масса:391.01
- Точка кипения:
- Номер Mdl:MFCD22690733
- Хранение:2-8°C
Category Infos
- Imidazoles
- Imidazole is an important five-membered nitrogen-containing heterocyclic compound. Among the numerous heterocyclic compounds, imidazole and its derivatives are regarded as a unique and multifaceted scaffold material due to their diverse applications in industrial, organic and pharmaceutical chemistry. Imidazoles interact in different ways with many therapeutic targets, enzymes and receptors in biological systems and thus exhibit a wide range of biological activities. In particular, several imidazoles can be used as clinical drugs to treat various types of cancer with high therapeutic efficacy. Furthermore, imidazoles are one of the most critical segments in the field of anti-covid-19 virus drug discovery due to their ability to interact with active targets in living systems.
- Imidazole Manufacturer
- As a professional imidazole manufacturer, we can guarantee product quality and delivery time.our company has been committed to supply personalized product solutions to customers,pls consult us.
Column Infos
- Inavolisib
- Genentech’s Phase III results show that the Inavolisib (GDC-0077) treatment meets the primary end point in hormone receptor (HR)-positive, HER2-negative, PIK3CA-mutated Breast Cancer. The combination of Inavolisib with Palbociclib and fulvestrant improves the progression-free survival (PFS) in the first line. Inavolisib is a potent selective inhibitor of PI3K alpha (PI3Kα) that binds to the ATP-binding site of p110α, and promotes the degradation of mutated p110α.